Genetic and environmental factors both clearly play a role in narcolepsy-cataplexy. Until now, the best evidence for autoimmunity as a cause of the disorder was the discovery that nearly everyone with the disorder has unique variants of a gene called HLA-DQB1*0602. Generally, though, both the chip data sets [ 1 , 2 ] are broadly consistent with previous findings. We can also compare the chip data with those from studies of immune-responsive genes in mosquito cells, which give many similar results, including differential regulation of large numbers of serpins (serine-protease inhibitors), pattern-recognition proteins, such as the peptidoglycan recognition protein (PGRP) family, and effectors involved in melanization and coagulation, such as prophenol oxidase [ 4 ].
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Cell walls have to allow the various nutrients that body cells need from the blood, but stop harmful pathogens. They must be stable.